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Complete OATP1B1 and OATP1B3 deficiency causes human Rotor syndrome by interrupting conjugated bilirubin reuptake into the liver

This aspect of ease of administration enhances the overall treatment experience, making it simpler and more reliable for patients to integrate into their health routines

In addition, we discovered that the AST released by RHEA@Gel could increase the expression of peroxidase and SOD in cells, thereby scavenging intracellular ROS

Another strategy is target-modification

BMC Cancer 15 , 856 (2015)

Es kommt natrlicherweise im menschlichen Blut und Gewebe vor und wird seit Jahrzehnten wissenschaftlich untersucht