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Efficient methylation of catechol estrogens is, therefore, of foremost importance: during methylation, a methyl group is donated by S-adenosylmethionine (SAM) via COMT to ensure that catechol estrogens are converted to methoxyestrogens, thereby limiting the formation of genotoxic estrogen quinones 18

It improves glucose homeostasis through various mechanisms including activation of insulin secretion and inhibition of glucagon secretion, delaying gastric emptying, reducing hepatic glucose production, and decreasing food intake

GLP-1 receptor agonists, are we witnessing the emergence of a paradigm shift for neuro-cardio-metabolic disorders?.Pharmacol Ther

Most patients need continued therapy to maintain benefits, though some may attempt supervised discontinuation after achieving significant metabolic improvements

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