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Consistent with an iron-sulfur cluster defect, SLC25A39/40 knock out cells displayed reduced activity of the enzyme aconitase, in which iron sulfur clusters function in both substrate binding and catalysis (Fig 4f)

At days 7 and 14 post-injury, the tensile breaking force portrayed higher values in the test subjects treated with a topical cream form of BPC 157

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In contrast, replacement with moderate physiological doses of GCs appears to prevent degenerative changes and support the survival and integration of newborn neurons in the DG

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