Despite initial enthusiasm for cuproptosis as a novel pathogenetic mechanism [27,51,54] , the molecular and metabolic complexities underlying copper-dependent hepatocellular injury in Wilson disease remain unresolved, especially since the role of ROS and reactive copper intermediates was not originally included in the cuproptosis framework [51,54]
SODs can be inhibited also by copper chelators such as EDTA and cuprizone (a copperspecific chelator) (Zhu et al
It works by neutralizing free radicals unstable molecules that can damage cells if left unchecked
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