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166 A recent study reported that S-nitrosylation altered AAT to a proinflammatory protein, which facilitated the inflammatory state of macrophages by upregulating proinflammatory molecules and the activation of MAPK signalling pathways

364 In addition, antileukemia activity has been observed in some AML patients

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Antioxidant mechanisms: Fenton reaction prevention through copper chelation, SOD upregulation, Nrf2 pathway activation, and lipid peroxidation quenching