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Atrial remodeling: (1) Structural remodeling: Multiple factors, including inflammatory responses, paracrine/vasocrine effects of EAT, and elevated chronic hemodynamic stress, can activate atrial fibroblasts, promote their proliferation and differentiation, alter the expression of relevant proteins and cytokines, and ultimately drive the pathogenesis and progression of atrial fibrosis (4045)

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In order to induce T lymphocyte apoptosis, GSH must be pumped out of the cells (Franco and Cidlowski, 2006, 2012

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people on stimulants, hormone modulators, or experimental metabolic compounds without clinician oversight