In these settings, GSH promotes cell death by enabling metal toxicity, sustaining redox-active metabolism, and amplifying oxidative or inflammatory signaling
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The same set of experiments was conducted for GSSSG-pretreated HGF as a control and results indicated a nearly negligible preventive-effect on HGF nitration (Figure 2c), consistent with the earlier observation that GSSSG treatment barely enhanced HGF-c-met interaction before and after exposure to peroxynitrite (see again Figure 1e, bars 47 and bars 47 )
The construct was transformed into E.coli Rosetta cells (AlpaLifeBio, KTSM106L) for protein expression
[DOI] [PubMed] [Google Scholar] Tong R, Wei C, Pan L, Zhang X (2020) Effects of dopamine on immune signaling pathway factors, phagocytosis and exocytosis in hemocytes of Litopenaeus vannamei
N-acetylcysteine and neurodegenerative diseases: Basic and clinical pharmacology
