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We observed that the dramatic increase in mitochondrial lipid peroxidation induced by LDHB silencing is not only associated with shrunken mitochondria featuring decreased crista and condensed/ruptured membranes, a phenotype associated with the induction of ferroptosis 28,29 but also with the accumulation of autophagosomes (Fig

Selectively reaching only the cancerous tissue remains one of the major challenges in RT [16]

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Collectively, these observations raise the possibility that MAMs function as platforms for integrating metabolic cues and hormonal signals during luteogenesis, a hypothesis that warrants direct experimental validation in ovarian models