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Such data may support hypotheses about endothelial response, vascular recruitment, and microvascular repair-associated mechanisms in defined laboratory models

Activation of GHS-R1a phospholipase C IP3 + DAG release of calcium from intracellular stores (endoplasmic reticulum) cytosolic calcium influx fusion of GH vesicles with the membrane exocytosis

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Enzymes that produce ROS, including ALOX, xanthine dehydrogenase, NADPH oxidase, and the mitochondrial electron transport chain complex, bind to iron, heme, or iron-sulfur clusters

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