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Notably, the affinity of cysteine for cysteinyl-tRNA synthetase is orders of magnitude higher than its affinity for the rate-limiting enzyme in GSH synthesis (GCLC) 48,49

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In HLU, both perform similarly because disuse atrophy involves both mTORC1 suppression (IGF-1 LR3 relevant) and myostatin upregulation (follistatin relevant) in approximately equal measure